Complement activity is regulated in C3 glomerulopathy by IgG–factor H fusion proteins with and without properdin targeting domains - ScienceDirect

Frontiers Modeling C3 glomerulopathies: C3 convertase regulation on an extracellular matrix surface

C3d-Targeted factor H inhibits tissue complement in disease models and reduces glomerular injury without affecting circulating complement - ScienceDirect

The role of the alternative pathway of complement activation in glomerular diseases

A C3-specific nanobody that blocks all three activation pathways in the human and murine complement system - ScienceDirect

C3 Glomerulopathy: Update on Pathogenesis and Treatment in: Kidney News Volume 13 Issue 4 (2021)

JCI - Human C3 glomerulopathy provides unique insights into complement factor H–related protein function

Monomeric CRP contributes to complement control in fluid phase and on cellular surfaces and increases phagocytosis by recruiting factor H

C3 glomerulopathies (C3G) are caused by dysregulation of the

Frontiers The role of the complement system in kidney glomerular capillary thrombosis

Complement inhibition in C3 glomerulopathy - ScienceDirect

Regulation of regulators: Role of the complement factor H-related proteins - ScienceDirect