GC−MS analysis of 786-O and isogenic SETD2-deficient 38E/38F

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Integrated Genomic and Proteomic Analyses Reveal Novel Mechanisms of the Methyltransferase SETD2 in Renal Cell Carcinoma Development - ScienceDirect

Simple and sensitive GC/MS-method for the quantification of urinary phenol, o- and m-cresol and ethylphenols as biomarkers of exposure to industrial solvents - ScienceDirect

PDF) Loss of SETD2 Induces a Metabolic Switch in Renal Cell Carcinoma Cell Lines toward Enhanced Oxidative Phosphorylation

EU Regulation Compliant GC-MS/MS Method Package for Dioxins in Foods - Features : Shimadzu Scientific Instruments

Hepatic lipid profile in the LDLR knockout rats. Lipids were extracted

Prkdc gene editing strategy and outcomes, ZFN design and gene

RNA-seq analysis of 786-O and isogenic SETD2-deficient 38E and 38F

SETD2 deficiency accelerates sphingomyelin accumulation and promotes the development of renal cancer

177 Lu-DOTATATE selectively killed hSSTr2 + ESCs in vitro. (A) hSSTr2 +

summarises engraftment, vector copy number (VCN), phenotypical analyses

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