The hypothesized role of miR-34a-5p in the IRE1α branch of UPR
CRISPR/Cas9-mediated inactivation of miR-34a and miR-34b/c in HCT116 colorectal cancer cells: comprehensive characterization after exposure to 5-FU reveals EMT and autophagy as key processes regulated by miR-34
Salicylate induces AMPK and inhibits c-MYC to activate a NRF2/ARE/miR-34a/b/ c cascade resulting in suppression of colorectal cancer metastasis
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Activation of miR-34a impairs autophagic flux and promotes cochlear cell death via repressing ATG9A: implications for age-related hearing loss
CRISPR/Cas9-mediated inactivation of miR-34a and miR-34b/c in HCT116 colorectal cancer cells: comprehensive characterization after exposure to 5-FU reveals EMT and autophagy as key processes regulated by miR-34
CRISPR/Cas9-mediated inactivation of miR-34a and miR-34b/c in HCT116 colorectal cancer cells: comprehensive characterization after exposure to 5-FU reveals EMT and autophagy as key processes regulated by miR-34
Gene set enrichment analysis of predicted miR-34a, miR-34b, and miR-34c
PDF] The Critical Role of Dysregulated FOXM1–PLAUR Signaling in Human Colon Cancer Progression and Metastasis
Hypoxia-induced IRE1A is repressed by miR-34a. (a) Western blot
Targeting the miR-34a/LRPPRC/MDR1 axis collapse the chemoresistance in P53 inactive colorectal cancer
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Salicylate induces AMPK and inhibits c-MYC to activate a NRF2/ARE/miR-34a/b/ c cascade resulting in suppression of colorectal cancer metastasis
Genetic determinants of FOXM1 overexpression in epithelial ovarian